gsa 300 microarray random priming labeling kit Search Results


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ATCC epithelial cell growth kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Epithelial Cell Growth Kit, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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epithelial cell growth kit - by Bioz Stars, 2026-08
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Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Aaf 1002x Miseq Reagent Kit V2 300 Cycles, supplied by Illumina Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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aaf 1002x miseq reagent kit v2 300 cycles - by Bioz Stars, 2026-08
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Qiagen rneasy mini kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Rneasy Mini Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Qiagen rneasy kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Rneasy Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Rneasy Minelute Cleanup Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Genisphere llc 3dna submicro ex expression array detection kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
3dna Submicro Ex Expression Array Detection Kit, supplied by Genisphere llc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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DIAGENODE DIAGNOSTICS high sensitivity dna chip
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
High Sensitivity Dna Chip, supplied by DIAGENODE DIAGNOSTICS, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Qiagen rneasy micro kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Rneasy Micro Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Qiagen rneasy plant mini kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
Rneasy Plant Mini Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Zymo Research chip dna clean concentrator kit
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
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Coherent Corp water cooled lasers
Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway <t>epithelial</t> cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B
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Image Search Results


Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway epithelial cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B

Journal: Cell Death & Disease

Article Title: Dual targeting of glutaminase 1 and thymidylate synthase elicits death synergistically in NSCLC

doi: 10.1038/cddis.2016.404

Figure Lengend Snippet: Glutamine metabolism is critical for overall survival and proliferation of NSCLC. ( a ) As was the case with the clinical information dataset from TCGA, higher levels of expression of GLS and TYMS were associated with reduced overall survival. Log-rank P -values of the genes were 0.0043 and 0.0135, respectively. Cases with higher expression levels of GLS and TYMS are coloured red, and cases without higher expression levels of these genes are coloured black. ( b ) Immunohistochemical staining of GLS1. The expression level of GLS1 in NSCLC tumour tissues was significantly higher than that in penumocytes from normal lung tissues ( N =57).The expression score was obtained from immuno-staining intensity and the percentage of positive cells in tissue microarray core as in Materials and Methods. The expression score was significantly higher in NSCLC tumor cells than in normal lung pneumocytes ( P <0.0001 by Mann-Whitney U test). ( c ) NSCLC cells were treated with glutamine-free medium for 48 h, and cell proliferation was tested by the SRB assay. ( d ) NSCLC cells were treated with GLS1 inhibitor, bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BPTES, 100 μM), for 48 h, and cell proliferation was tested by the SRB assay. ( e ) The expression level of GLS1 in NSCLC cells and the primary small airway epithelial cells IMR90 was analysed by immunoblotting. SRB, sulforhodamine B

Article Snippet: Lung primary cell was airway epithelial cell basal medium (PCS-300-030, ATCC, Manassas, VA, USA) with the bronchial epithelial cell growth kit (PCS-300-040, ATCC, Manassas, VA, USA).

Techniques: Expressing, Immunohistochemical staining, Staining, Immunostaining, Microarray, MANN-WHITNEY, Sulforhodamine B Assay, Western Blot